Serveur d'exploration sur le patient édenté

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Urban legends series: oral candidosis

Identifieur interne : 002804 ( Main/Exploration ); précédent : 002803; suivant : 002805

Urban legends series: oral candidosis

Auteurs : M. Manfredi [Italie] ; L. Polonelli [Italie] ; Jm Aguirre-Urizar [Espagne] ; M. Carrozzo [Royaume-Uni] ; Mj Mccullough [Australie]

Source :

RBID : ISTEX:4F1AE4227B2CB9F322F5FDC4193A1123531FDE6F

English descriptors

Abstract

Candida species (spp) are commensal yeast that can only instigate oral infection (oral candidosis – OC) when there is an underlying predisposing condition in the host. We investigated four controversial topics on OC: (i) How can a microbiological determination of OC be made as Candida spp. are commensal yeasts and not all of them form hyphae or pseudohyphae during infection? (ii) Is median rhomboid glossitis (MRG) a manifestation of candidal infection? (iii) Can candidal infection cause palate papillary hyperplasia (PPH)? (iv) What is the best therapeutic treatment for denture‐associated erythematous stomatitis (DAES)? Results from extensive literature searches, including a systematic review, suggested the following: (i) the diagnosis of OC merely on the basis of the presence of yeasts is an oversimplification of a complex process. No convincing evidence of a single test or method better able to discriminate the transition from candidal saprophytism to pathogenicity has been reported in the literature; (ii–iii) conclusive evidence of a direct aetiopathogenic relationship between MRG and PPH and candidal infection has not been found; and (iv) only limited evidence is available for any DAES treatment, thus making it impossible to make strong therapeutic recommendations.

Url:
DOI: 10.1111/odi.12013


Affiliations:


Links toward previous steps (curation, corpus...)


Le document en format XML

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<term>Antifungal treatment</term>
<term>Arendorf</term>
<term>Aust dent</term>
<term>Biol</term>
<term>Biol buccale</term>
<term>Candida</term>
<term>Candida albicans</term>
<term>Candida species</term>
<term>Candida strains</term>
<term>Candidal</term>
<term>Candidal infection</term>
<term>Candidiasis</term>
<term>Candidosis</term>
<term>Chlorhexidine</term>
<term>Cleanser</term>
<term>Clin</term>
<term>Clinical evaluation</term>
<term>Clinical signs</term>
<term>Cochrane</term>
<term>Colonisation</term>
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<term>Commensal yeasts</term>
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<term>Complete dentures</term>
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<term>Daes treatment</term>
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<term>Denture</term>
<term>Denture cleanser</term>
<term>Denture cleansers</term>
<term>Denture stomatitis</term>
<term>Epstein</term>
<term>Erythematous</term>
<term>Erythematous candidosis</term>
<term>Farah</term>
<term>Form hyphae</term>
<term>Fungal</term>
<term>Fungal hyphae</term>
<term>Genus candida</term>
<term>Glossitis</term>
<term>Hyperplasia</term>
<term>Hyperplastic</term>
<term>Hypha</term>
<term>Immunocompromised patients</term>
<term>Infection</term>
<term>Itraconazole</term>
<term>Lamentous forms</term>
<term>Laser</term>
<term>Lesion</term>
<term>Manfredi</term>
<term>Median</term>
<term>Median rhomboid glossitis</term>
<term>Miconazole</term>
<term>Miconazole lacquer</term>
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<term>Microbiological</term>
<term>Microbiological determination</term>
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<term>Systematic review</term>
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<term>Adverse reactions</term>
<term>Albicans</term>
<term>Antifungal</term>
<term>Antifungal treatment</term>
<term>Arendorf</term>
<term>Aust dent</term>
<term>Biol</term>
<term>Biol buccale</term>
<term>Candida</term>
<term>Candida albicans</term>
<term>Candida species</term>
<term>Candida strains</term>
<term>Candidal</term>
<term>Candidal infection</term>
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<term>Commensal yeasts</term>
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<term>Complete dentures</term>
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<term>Fungal hyphae</term>
<term>Genus candida</term>
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<term>Hyperplasia</term>
<term>Hyperplastic</term>
<term>Hypha</term>
<term>Immunocompromised patients</term>
<term>Infection</term>
<term>Itraconazole</term>
<term>Lamentous forms</term>
<term>Laser</term>
<term>Lesion</term>
<term>Manfredi</term>
<term>Median</term>
<term>Median rhomboid glossitis</term>
<term>Miconazole</term>
<term>Miconazole lacquer</term>
<term>Microbiol</term>
<term>Microbiological</term>
<term>Microbiological determination</term>
<term>Mouth rinses</term>
<term>Mouthwash</term>
<term>Mucosal</term>
<term>Mycological</term>
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<term>Oral candida</term>
<term>Oral candidiasis</term>
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<term>Pseudohyphae</term>
<term>Punica granatum</term>
<term>Pure culture</term>
<term>Radiol endod</term>
<term>Randomisation</term>
<term>Randomisation method</term>
<term>Rcts</term>
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<term>Salivary</term>
<term>Samaranayake</term>
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<term>Silverman</term>
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<front>
<div type="abstract">Candida species (spp) are commensal yeast that can only instigate oral infection (oral candidosis – OC) when there is an underlying predisposing condition in the host. We investigated four controversial topics on OC: (i) How can a microbiological determination of OC be made as Candida spp. are commensal yeasts and not all of them form hyphae or pseudohyphae during infection? (ii) Is median rhomboid glossitis (MRG) a manifestation of candidal infection? (iii) Can candidal infection cause palate papillary hyperplasia (PPH)? (iv) What is the best therapeutic treatment for denture‐associated erythematous stomatitis (DAES)? Results from extensive literature searches, including a systematic review, suggested the following: (i) the diagnosis of OC merely on the basis of the presence of yeasts is an oversimplification of a complex process. No convincing evidence of a single test or method better able to discriminate the transition from candidal saprophytism to pathogenicity has been reported in the literature; (ii–iii) conclusive evidence of a direct aetiopathogenic relationship between MRG and PPH and candidal infection has not been found; and (iv) only limited evidence is available for any DAES treatment, thus making it impossible to make strong therapeutic recommendations.</div>
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